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Target
Application
Host
Type
Species Reactivity
Expression Host
Conjugate
Derivation
INHA
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For Research Use Only. Not For Clinical Use.
Background
inhA, or Enoyl-[acyl-carrier-protein] reductase [NADH], is an enzyme in the fatty acid biosynthesis pathway of Mycobacterium tuberculosis. It catalyzes the NADH-dependent reduction of enoyl-ACP, a crucial step for producing long-chain fatty acids essential for mycobacterial cell wall synthesis. InhA is the target of the antitubercular drug isoniazid.
Predicted location
Secreted
Single cell type specificity
Cell type enhanced (Syncytiotrophoblasts, Granulosa cells, Theca cells)
Immune cell specificity
Not detected in immune cells
Cell line specificity
Cell line enhanced (BEWO, HEL, SK-BR-3, SuSa)
Interaction
Dimeric, linked by one or more disulfide bonds. Inhibin A is a dimer of alpha and beta-A. Inhibin B is a dimer of alpha and beta-B.
Molecular function
Growth factor, Hormone
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