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AML
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- Recombinant Anti-human AML Antibody (MOB-60)
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- Derivation: Mouse
- Species Reactivity: Human
- Type: IgG
- Application: WB, Dot, FuncS
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- Recombinant Anti-human AML Antibody Fab Fragment (MOB-60-F(E))
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- Derivation: Mouse
- Species Reactivity: Human
- Type: Fab
- Application: Neut, WB, FuncS
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- Recombinant Anti-human AML Antibody scFv Fragment (MOB-60-S(P))
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- Derivation: Mouse
- Species Reactivity: Human
- Type: scFv
- Application: RIA, ELISA, FuncS
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- Recombinant Human Anti-human AML Antibody Fab Fragment (MHH-60-F(E))
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- Derivation: Human
- Species Reactivity: Human
- Type: Fab
- Application: WB, FC, FuncS
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- Recombinant Human Anti-human AML Antibody scFv Fragment (MHH-60-S(P))
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- Derivation: Human
- Species Reactivity: Human
- Type: scFv
- Application: Neut, IF, FuncS
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- Mouse Anti-AML scFv-Fc Chimera (VS-0125-FY111) (VS-0125-FY111)
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- Species Reactivity: Human
- Type: Mouse IgG2a, scFv-Fc
- Application: BI
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For Research Use Only. Not For Clinical Use.
Background
AML (Acute Myeloid Leukemia) protein, specifically the RUNX1 gene, plays a vital role in hematopoiesis and is essential for the regulation of blood cell development. Mutations and rearrangements in the RUNX1 gene are commonly found in various blood disorders, including acute myeloid leukemia (AML), thereby linking this gene to cancer pathogenesis.